Objectives

Further understand the mechanism and potential clinical applications of biofield therapy for pancreatic cancer

Hypotheses

• Biofield therapy causes shifts in cell-level bioelectric shifts which precede the biochemical cascade of effects observed mechanistically invitro and invivo
• Preclinical research on biofield therapy for pancreatic cancer will show tumor microenvironment shifts, specifically immune cell landscape shifts, across a variety of cancers, animals, dosage levels, and in combination with conventional therapies

Expected Outcomes (In Process)

• Develop a cell cycle-level understanding by substage of the antimetastatic effects of biofield therapy
• Confirm whether biofield therapy has the ability to modulate cell-level bioelectricity
• Determine whether cell-level bioelectric shifts lead to the biochemical shifts observed
• Evaluate new potential clinical delivery methods